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Anti-apoptotic protection of dermal papilla cells AHK-Cu protected DPCs from programmed cell death through multiple molecular pathways: Bcl-2/Bax rebalancing: Bcl-2 (an anti-apoptotic protein dominant during the telogen-to-anagen transition) was upregulated, while Bax (a pro-apoptotic protein) was downregulated

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Additionally, many of the currently available clinical agents display the always on characteristic, where they illuminate disease sites through self-accumulation rather than being specifically activated by target molecules of interest, which leads to poor imaging contrast and compromised detection accuracy

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