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Mitochondrial function is required for extracellular ATP-induced NLRP3= inflammasome activation

These results show that Pep-B has anti-inflammatory activity and promotes osteo-/odontoblastic differentiation in the LPS-induced inflammatory environment and may have a potential role in hDPSCs for repair and regeneration [31]

GLP-1RAs reduce the accumulation of advanced glycation end-products (AGEs), which bind to the receptor for AGEs (RAGE), and trigger pro-inflammatory pathways that contribute to lung pathology [130,131,132,133,134]