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These changes may compromise blood-brain barrier integrity, promote oxidative stress, and initiate a feedback loop that exacerbates neuroinflammation (Bandyopadhyay & Shukla, 2023)

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The kidney-protective properties of LEO against cell death appear to be mediated through diminished NLRP3 and inflammatory mediators, improving mitochondrial function, and attenuating endoplasmic reticulum (ER) stress