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In the ALS rodent model, the mitochondrial biogenesis regulator PGC-1 has been demonstrated to protect upper motor neurons, participate in the production of neuromuscular junctions in lower motor neurons, and regulate oxidative stress in sensory neurons (Kuczynska et al., 2021)

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Its primary mechanism involves the upregulation of growth factor receptors and the promotion of angiogenesis (the formation of new blood vessels), which are critical components of the tissue repair process