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doi: 10.1016/j.ajhg.2008.04.019 31 TaylorPNPanickerVSayersAShieldsBIqbalABremnerAPet al

Regarding mitochondrial-mediated apoptosis, since cardiomyocytes have a significant vulnerability to the ischemic episode, the prolongation of MI leads to the significant disruption of cardiac architecture and function by impairing mitochondrial dynamics and function 21,23

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