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Furthermore, knocking down CBS resulted in a 10-fold induction of a master regulator of EMT, SNAI2 (SLUG) (Supplementary Fig

Although additional studies are required to clarify the molecular mechanisms responsible for hyperhomocysteinemia-induced vascular disorders, there is evidence that the direct cytotoxicity of markedly elevated homocysteine and/or of its oxidized by-products (homocysteine thiolactone, homocysteine sulfinic acid, and homocysteic acid) damages endothelial cells (Dudman et al, 1991

Zachara BA, Dobrzyski W, Trafikowska U, Szymaski W

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