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Cloning and characterization of inducible nitric oxide synthase from mouse macrophages

To understand the molecular mechanisms underlying dysregulation of SAA metabolism during liver cancer development, we first investigated the relationship between the expression levels of five well-established hepatic HNF4 target genes, five general mesenchymal markers, and eight SAA metabolic enzymes in the TCGA LIHC dataset of 373 HCC patients

Aging is considered a precipitating factor for both increased oxidative stress [114] and decreased GSH levels in the brain [115]

Thioredoxin reductase 1 (TXNRD1) is predominantly located in the cytoplasmic lysis and contains a catalytically essential selenocysteine residue at its C-terminal active site