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In all patterns of ethanol consumption described above, we reported changes associated with mitochondrial injury [11, 27, 132], therefore strongly suggesting that mitochondria is an important mediator of ethanol neurotoxicity and could be considered as a potential therapeutic approaches for treating ethanol-associated disorders

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Abbreviation: apoA1 apolipoprotein A1, apoB apolipoprotein B, lp(a) lipoprotein (a), HDL-c High density lipoprotein cholesterol, LDL-c Low density lipoprotein cholesterol, TC Total cholesterol