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AKT phosphorylation inhibits Tuberous sclerosis protein 2 (TSC2), a GTPase activating protein (GAP), leading to activation of mTOR1 complex followed by activation of S6K

The potential therapeutic benefits of blocking A 2A receptor signaling in microglia appear broadly applicable across neurodegenerative diseases

I.PappA.GyngyiZ.FrizsI.et al (2022)

158 Available findings have allowed us to speculate that the biological activities of oxidized AAT may depend on the magnitude of AAT oxidation, in a manner similar to that discussed for HSA (Figure 3)