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poor nutrition, environmental toxins, and stress

Results Neuronal GCLC knockout leads to brain atrophy accompanied by neuronal cell death To examine how the declines in GCLC and glutathione might affect brain homeostasis, we prepared GCLC floxed X CaMKII-Cre (GCLC-KO) mice whose GCLC expression is deleted in calcium/calmodulin-dependent protein kinase II (CaMKII)-positive neurons

In addition, oxidative stress can also trigger DNA damage that leads to genomic instability and modification of the cellular mutational status of AML cells, thus favoring tumor progression [34]

These responses including glial activation, metabolic braking, and suppression of plasticity-related signaling are biologically adaptive in the short term, but may become limiting when they remain chronically engaged