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Considering the low yield of expressed protein with pAcF incorporated, we engineered pCNFRS to improve the incorporation of pAcF by introducing a quadruple mutation H283T/P284S/M285D/D286V (TSDV) located at the anticodon recognition domain that was proved useful in pAzFRS for improving the incorporation of (p-azido-phenylalanine) pAzF 28

To gain insights into TBI pathogenesis, we developed a novel murine closed-skull brain injury model that mirrors some pathological features associated with mild TBI in humans and used long-term intravital microscopy to study the dynamics of the injury response from its inception

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Mace TA, Ameen Z, Collins A, Wojcik S, Mair M, Young GS, et al