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However, Aja and colleagues found no evidence for this hypothesis since in this model CPT-1 should inhibit feeding by increasing cytosolic fatty-acyl CoA levels while they actually showed the initial response of mice to a CPT-1 inhibitor was an increase in appetite [75]

He has experience in planning, managing, and supervising biomedical research projects, with expertise in cellular and molecular biology, redox biology, biochemistry, mammalian cell culture, and endocrine physiology.

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