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Indeed, the C677T mutation of the MTHFR gene is not unequivocally associated with increased cardiovascular risk

RSL3 (0.1 M) treatment increased -H2AX levels and previously unreported reduced pNPM1 (T199) levels when compared to control treatment

Based on clinical patterns and research shared in my articles like "High Homocysteine, MTHFR Mutations, and the Inflammation Crisis," here are the major risks: Chronic Inflammation and Pain : Elevated homocysteine ramps up cytokines like IL-8 (neutrophil recruiter) and MCP-1 (monocyte attractor), leading to widespread aches, muscle tenderness, and "thermal runaway" inflammation

Heatmap showing fold change in metabolite levels (log 2 ) of the indicated cell lines relative to the average of those in SLC25A39 knockout cells complemented with SLC25A39 cDNA (middle)